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The concept of “positive selection” (PS) arises from the need to understand evolutionary mechanisms leading to species differences or the adaptation to a given environment. This research aimed to establish whether the human body harbours potential signals of PS. Authors hypothesised that molecular phylogenies reconstructed in the human genome based on intronic sites have a better statistical support than human-chimpanzee orthologous genes. These phylogenies have then been used to explore the incidence of PS in comparison with species pairs defining the human and the non-human primate (NHP) clades, as well as with the NHP species that in our sample of the human genome span a large geographical and taxonomic range. The main results of the study were: first, phylogenies based on intronic sites of intronic-only genes have a better statistical support (as measured by the SH-like tree support) than phylogenies based on intronic and coding sites for human and NHP comparisons (PUpregulation of L-type calcium current in K562 cells by the activation of protein kinase C and induction of the acute promyelocytic leukemia-associated fusion protein, PLZF.
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